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Publishedgenerated 2026-05-30T11:25:54Z

T-VEC at antigen release, addressing threshold drift

T-VEC (oncolytic lysis with GM CSF) acts at the antigen release step of the cancer-immunity cycle. The failure mode is threshold drift: threshold moves out of reach of normal loading current. Capacitor sometimes fires, sometimes doesn't; unpredictable activation.

C4·I3
composite 0.51structural 0.80evidence 0.24novelty 0.50

What this is

The Living Scorecard is Encounter's open record of structural predictions. Each hypothesis names a candidate intervention, where in the cycle it is predicted to act, the failure mode it is predicted to address, and the evidence available when the prediction was made.

Predictions are locked with timestamps before they can be tested. Encounter generates them daily, many more than any one person can chase, so the field can see what the framework claims structurally, ahead of the data. Some will hold. Some will be refuted. Both stay on the page. The point is that each prediction is committed before the answer is known, so the data that arrives later can confirm or refute it.

Structural argument

At the antigen release step of the cancer-immunity cycle (capacitor regime, position P1), T-VEC acts via oncolytic lysis with GM CSF. The failure mode is threshold drift: threshold moves out of reach of normal loading current. Capacitor sometimes fires, sometimes doesn't; unpredictable activation. The intervention's regime-type class lines up with the position's regime, and failure_threshold_drift belongs to that regime's failure set.

  • rule_r13 (required) — Combinatorial candidate is grounded in R13's position-to-state table, which determines which interventions are structurally addressable at which positions.

Substrate

Domain
domain_cellular_biology
Cycle
cycle_l4_canonical
Position(s)
position_p1
Failure modes
failure_threshold_drift

Evidence at generation

PubMed35 hits

"t-vec" AND "cancer immunotherapy"

  • PMID 30643254 (2019) Nat Genet

    Tumor mutational load predicts survival after immunotherapy across multiple cancer types.

  • PMID 33803762 (2021) Cancers (Basel)

    Talimogene Laherparepvec (T-VEC): An Intralesional Cancer Immunotherapy for Advanced Melanoma.

  • PMID 26488034 (2015) Drugs Today (Barc)

    Talimogene laherparepvec (T-VEC) as cancer immunotherapy.

  • PMID 30832995 (2019) Hematol Oncol Clin North Am

    Cancer Vaccines.

  • PMID 40043281 (2025) Expert Rev Anticancer Ther

    Melanoma neoadjuvant treatment: review and update of recent trials.

ChEMBLNo mechanism match

Targets:

Not found in ChEMBL: T-VEC (talimogene laherparepvec)

ClinicalTrials.gov5 trials

t-vec cancer immunotherapy

  • NCT01740297PHASE1,PHASE2 · COMPLETED

    Melanoma

    Primary: Phase 1b: Number of Participants With Dose-limiting Toxicities

  • NCT04163952PHASE1 · TERMINATED

    Locally Advanced Skin Squamous Cell Carcinoma

    Primary: Number of Participants With Treatment Related Adverse Events as Assessed by CTCAE v4.0

  • NCT03300544PHASE1 · TERMINATED

    Locally Advanced Rectal Adenocarcinoma

    Primary: Determination of Dose Limiting Toxicities (DLTs) and Maximum Tolerated Dose (MTD) of Talimogene Laherparepvec in Combination With Chemotherapy and Radiation in Rectal Cancer.

  • NCT03842943PHASE2 · RECRUITING

    Cutaneous Melanoma

    Primary: Pathologic Complete Response

  • NCT03071094PHASE1,PHASE2 · TERMINATED

    Hepatocellular Carcinoma (HCC)

    Primary: Phase I: Number of Participants With Dose Limiting Toxicities (DLTs)

Status timeline

  • Draft2026-05-30T11:27:14Z

    Generated by combinatorial walker (v0.1.1; verb_target-aware, sub-regime failures). Awaiting operator review for the daily top-5 batch.

    by system

Tags

confidence:4impact:3generation:combinatorialnote:top5-batch